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- W195243966 abstract "Restenosis following angioplasty traditionally has been attributed to intimai hyperplasia involving the release of growth factors. Growth factors presumably induce a subgroup of smooth muscle cells in the media of the vessel wall to undergo phenotypic transformation, migrate into the intima, proliferate and secrete extracellular matrix [1]. A new concept that has been proposed recently is that constrictive remodeling with a reduction in vessel caliber may be another mechanism that contributes to restenosis [2, 3].KeywordsSmooth Muscle CellChronic HypertensionResistance ArterySmall Mesenteric ArteryCerebral ArterioleThese keywords were added by machine and not by the authors. This process is experimental and the keywords may be updated as the learning algorithm improves." @default.
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- W195243966 date "1997-01-01" @default.
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- W195243966 title "Constrictive Remodeling: Do Concepts of Remodeling During Chronic Hypertension Apply to Restenosis?" @default.
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