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- W1954559244 abstract "BRAF mutations are one of the many oncogenic drivers described in lung adenocarcinomas and are seen in approximately 3% of cases. The most common BRAF mutation corresponds to a hotspot transversion mutation T1799A at exon 15, which causes a valine to glutamine substitution at residue 600. The other BRAF mutations, classified as non-V600E, are distributed in exons 15 and 11, the most common being G469A in exon 11 (39%). The BRAF inhibitors vemurafenib and dabrafenib, specifically target the BRAFV600E mutant kinase. In this chapter the biology, molecular pathogenesis and clinical characteristics of BRAF mutated lung adenocarcinomas are described." @default.
- W1954559244 created "2016-06-24" @default.
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- W1954559244 date "2014-04-04" @default.
- W1954559244 modified "2023-10-16" @default.
- W1954559244 title "Non‐Small Cell Lung Cancers (NSCLC) with Mutations in <i>BRAF</i>" @default.
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- W1954559244 doi "https://doi.org/10.1002/9781118468791.ch36" @default.
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