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- W1957003034 endingPage "137" @default.
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- W1957003034 abstract "The mucosal layer of the gastrointestinal (GI) tract is able to resist digestion by the endogenous substances that we secrete to digest foodstuffs. So-called mucosal defense is multi-factorial and can be modulated by a wide range of substances, many of which are classically regarded as inflammatory mediators. Damage to the GI mucosa, and its subsequent repair, are also modulated by various inflammatory mediators. In this article, we provide a review of some of the key inflammatory mediators that modulate GI mucosal defense, injury, and repair. Among the mediators discussed are nitric oxide, polyamines, the eicosanoids (prostaglandins and lipoxins), protease-activated receptors, and cytokines. Many of these endogenous factors, or the enzymes involved in their synthesis, are considered potential therapeutic targets for the treatment of diseases of the digestive tract that are characterized by inflammation and ulceration." @default.
- W1957003034 created "2016-06-24" @default.
- W1957003034 creator A5027937626 @default.
- W1957003034 creator A5084326812 @default.
- W1957003034 date "2006-02-01" @default.
- W1957003034 modified "2023-10-01" @default.
- W1957003034 title "Gastrointestinal Inflammation: A Central Component of Mucosal Defense and Repair" @default.
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- W1957003034 doi "https://doi.org/10.1177/153537020623100202" @default.
- W1957003034 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/16446488" @default.
- W1957003034 hasPublicationYear "2006" @default.
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