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• W1959250619 endingPage "2616" @default.
• W1959250619 startingPage "2610" @default.
• W1959250619 abstract "The decrease in NK cell activity and the loss of gammadelta T cells in active pulmonary tuberculosis patients have been reported. In this study, we observed that the proliferating response of gammadelta T cells to the heat-treated Ags of Mycobacterium tuberculosis from different individuals was noted to be dependent on the content or function of NK cells in PBMC in a population study. We also found that NK cells were directly rapidly activated by the heat-treated Ags from M. tuberculosis (H37Ra) in vitro; in turn, the activated NK cells improved gammadelta T cell proliferation both by CD54-mediated cell-cell contact through the forming immune synapse and by soluble factors TNF-alpha, GM-CSF, and IL-12, but not IFN-gamma. Our results demonstrated that an interaction between NK cells and gammadelta T cells existed in antituberculosis immunity. Up-regulating the function of NK cells might be beneficial to the prevention and control of pulmonary tuberculosis." @default.
• W1959250619 created "2016-06-24" @default.
• W1959250619 creator A5012198733 @default.
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• W1959250619 date "2006-02-15" @default.
• W1959250619 modified "2023-09-26" @default.
• W1959250619 title "Human NK Cells Positively Regulate γδ T Cells in Response to <i>Mycobacterium tuberculosis</i>" @default.
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• W1959250619 doi "https://doi.org/10.4049/jimmunol.176.4.2610" @default.
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