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- W196247046 abstract "This chapter presents extracellular matrix interactions with tumor-progressing cells and their mechanism of actions. It presents a review of fibronectin (FN) adhesion mechanisms for only two tumor-cell systems. This indicates the complexity by which tumor cells interact with various matrices that they would encounter during tumor progression. Tumor cells can regulate adhesion processes at the level of matrix receptor gene expression and the nature and degrees of splicing of FN pre-mRNAs. The results analyzed in the chapter provide an insight into cell type-specific adhesion mechanisms, as differentiated from commonly shared tumor-specific mechanisms. In addition to transformant-associated changes in the amounts or catabolism of various integrin receptors, there are also cell type-specific differences in the catabolism of membrane-intercalated heparin sulfate proteoglycans in the substratum adhesion sites of these two tumors. Both neuroblastoma and ras/3T3 tumor cells utilize some degree of cooperativity between the cell t domain of FNs and a second domain cell x . Many more tumor cell systems should be analyzed, both in vivo and in vitro , during tumor progression, for their matrix adhesion mechanisms." @default.
- W196247046 created "2016-06-24" @default.
- W196247046 creator A5004059972 @default.
- W196247046 creator A5011929380 @default.
- W196247046 creator A5078787495 @default.
- W196247046 date "1991-01-01" @default.
- W196247046 modified "2023-09-26" @default.
- W196247046 title "Extracellular Matrix Interactions with Tumor-Progressing Cells: Tumor versus Cell Type-Specific Mechanisms" @default.
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