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• W1963481034 abstract "The role of CD8(+) T cells in oral tolerance remains unclear. To address this, we developed a model to induce CD8(+) Tregs by feeding the major histocompatibility complex class I immunodominant epitope of OVA, OVA((257-264)). OVA((257-264)) feeding induced tolerance similar to that observed in OVA protein-fed mice, capable of suppressing the production of Th1 and Th17 cytokines and inhibiting a Th1-driven delayed-type hypersensitivity response following immunization with whole OVA (wOVA) protein. OVA((257-264)) peptide-induced suppression could be transferred to naive mice with CD8(+) cells, but not CD8-depleted cells, isolated from mesenteric lymph nodes of peptide-fed mice. Interestingly, while capable of inhibiting Th1 and Th17 responses, OVA((257-264)) feeding could not suppress any feature of a Th2 inflammatory response, though OVA protein feeding could, suggesting that these cells function through a different mechanism than their CD4(+) counterparts generated in response to feeding with wOVA. Thus, CD8(+) T cells are functionally capable of mediating tolerance to Th1 and Th17 responses." @default.
• W1963481034 created "2016-06-24" @default.
• W1963481034 creator A5007095938 @default.
• W1963481034 creator A5044711082 @default.
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• W1963481034 date "2009-09-01" @default.
• W1963481034 modified "2023-10-11" @default.
• W1963481034 title "Suppression of Th1 and Th17, but not Th2, responses in a CD8+ T cell-mediated model of oral tolerance" @default.
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• W1963481034 doi "https://doi.org/10.1038/mi.2009.93" @default.
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