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• W1963999832 abstract "Alcohol consumption often diminishes antigen‐specific cell‐mediated immunity. In alcohol‐consuming mice IFN‐γ and delayed‐type hypersensitivity (DTH) responses are blunted, although antigen‐specific T cell proliferation and IL‐2 responses are largely unaffected, suggesting that alcohol differentially affects signal transduction pathways. In the present report we explore the use of the phosphatase inhibitor, Na3VO4 to restore IFN‐γ secretion in the presence of ethanol both in vivo and in vitro. We show that Na3VO4 restores IFN‐γ in vitro and antigen‐specific DTH in vivo to the levels seen in alcohol non‐consuming mice. Our data support the contention that ethanol, by up‐regulating phosphotyrosine phosphatase, diminishes the IFN‐γ signal transduction pathway." @default.
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• W1963999832 date "2002-11-01" @default.
• W1963999832 modified "2023-10-11" @default.
• W1963999832 title "Restoration of ethanol-compromised Th1 responses by sodium orthovanadate" @default.
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• W1963999832 doi "https://doi.org/10.1093/intimm/dxf088" @default.
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