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- W1964184147 abstract "To the Editor.— There is a sense of deja vu about the description by Bove et al 1 of the adverse reactions and deaths that followed the intravenous administration of the vitamin E preparation E-Ferol to low-birth-weight infants. The syndrome of renal dysfunction, hepatic dysfunction, metabolic acidosis, and tissue oxalosis has many similarities to that which occurs in patients who received parenterally large amounts of xylitol. 2 When first observed, the xylitol toxicity syndrome was unexpected, and, indeed, it was concluded that the oxalosis was not causally related to the infusion of xylitol. 2 However, both animal and biochemical studies have since confirmed that oxalate or oxalate precursors can be formed during the hepatic metabolism of xylitol, 2 with the enzymes fructokinase and aldolase playing important roles in the metabolic production of the oxalate precursor glycolaldehyde from xylitol. 2,3 In terms of a one-compartment model for the renal elimination of" @default.
- W1964184147 created "2016-06-24" @default.
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- W1964184147 date "1986-11-21" @default.
- W1964184147 modified "2023-10-18" @default.
- W1964184147 title "Oxalosis and the E-Ferol Toxicity Syndrome" @default.
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- W1964184147 doi "https://doi.org/10.1001/jama.1986.03380190047021" @default.
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