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- W1964479886 abstract "Rheumatoid arthritis (RA) has no firm etiologic basis. It progresses as anautoimmune disease and evolves into a chronic inflammatory joint disease complicated by recurrent episodes of systemic acute-phase reactions, which sometimes result in amyloidosis. Cytokines play a pivotol role in inflammation and the immune response. Proinflammatory cytokines such as interleukin-1, tumor necrosis factor α (TNF-α), and interleukin-6 are present at high levels in arthritic joints, and their blood concentration correlates with the severity of the RA. Some of the activities of the proinflammatory cytokines, such as stimulation of leukocyte infiltration and release of their proteolytic enzymes, may be mediated by acute phase proteins (APPS), such as C-reactive protein and serum amyloid A, and by chemokines such as interleukin-8. Cytokines, chemokines, and APPs reciprocally regulate each others' expression and activities, constituting a communication network between fibroblasts, macrophages, lymphocytes, and hepatocytes. Activation of the network results in inflammation and the progressive destruction of joints and systemic symptoms characteristic of RA." @default.
- W1964479886 created "2016-06-24" @default.
- W1964479886 creator A5042240287 @default.
- W1964479886 creator A5068327775 @default.
- W1964479886 date "1996-10-01" @default.
- W1964479886 modified "2023-10-18" @default.
- W1964479886 title "Role of cytokines, acute-phase proteins, and chemokinesin the progression of rheumatoid arthritis" @default.
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