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- W1965594668 endingPage "110" @default.
- W1965594668 startingPage "102" @default.
- W1965594668 abstract "Huntington's disease (HD) is a neurodegenerative disorder with an autosomal dominant expression pattern and typically a late-onset appearance. HD is a movement disorder with a heterogeneous phenotype characterized by involuntary dance-like gait, bioenergetic deficits, motor impairment, and cognitive and psychiatric deficits. Compelling evidence suggests that increased oxidative stress and mitochondrial dysfunction may underlie HD pathogenesis. However, the exact mechanisms underlying mutant huntingtin-induced neurological toxicity remain unclear. The objective of this paper is to review recent literature regarding the role of oxidative DNA damage in mitochondrial dysfunction and HD pathogenesis." @default.
- W1965594668 created "2016-06-24" @default.
- W1965594668 creator A5002884817 @default.
- W1965594668 date "2013-09-01" @default.
- W1965594668 modified "2023-09-26" @default.
- W1965594668 title "Role of oxidative DNA damage in mitochondrial dysfunction and Huntington’s disease pathogenesis" @default.
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