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• W1965816191 abstract "HomeCirculation ResearchVol. 94, No. 8Bone Marrow–Derived Cells and Vascular Growth Free AccessLetterPDF/EPUBAboutView PDFView EPUBSections ToolsAdd to favoritesDownload citationsTrack citationsPermissions ShareShare onFacebookTwitterLinked InMendeleyReddit Jump toFree AccessLetterPDF/EPUBBone Marrow–Derived Cells and Vascular Growth Dirk Skowasch, Alexander Jabs, Berndt Lüderitz and Gerhard Bauriedel Dirk SkowaschDirk Skowasch Department of Cardiology, University of Bonn, Bonn, Germany, Search for more papers by this author , Alexander JabsAlexander Jabs Department of Cardiology, University of Bonn, Bonn, Germany, Search for more papers by this author , Berndt LüderitzBerndt Lüderitz Department of Cardiology, University of Bonn, Bonn, Germany, Search for more papers by this author and Gerhard BauriedelGerhard Bauriedel Department of Cardiology, University of Bonn, Bonn, Germany, Search for more papers by this author Originally published30 Apr 2004https://doi.org/10.1161/01.RES.0000126418.31311.F8Circulation Research. 2004;94:e71To the Editor:We read with great interest the study by Ziegelhoeffer et al,1 who tested whether circulating bone marrow–derived cells incorporate into collateral arteries after femoral artery ligation. Although the investigators did not find any incorporation of bone marrow–derived cells in the endothelium and tunica media of growing vessels, they observed accumulations of bone marrow–derived cells in areas of collateral artery growth and capillary growth and identified these cells as fibroblasts, pericytes, and primarily leukocytes. These exciting data add another piece of evidence to the concept that apparently not mural, tissue-resident cells but other cell types play a more central role in vascular repair.Given the diversity of blood leukocyte populations, the exact identity of these cells deserves careful consideration. In this context, we recently identified dendritic cells as a novel neointimal cell type and demonstrated their maximal frequency during early neointima formation after rat carotid balloon injury.2 In another study, we also showed bone marrow– and neural crest–derived cells within human in-stent restenosis atherectomy samples.3 Herein, dendritic cells constituted the major mononuclear lesional cell type, whereas monocytes/macrophages were confined to areas adjacent to stent struts.3 It is tempting to speculate that dendritic cells may have contributed to the bone marrow–derived perivascular leukocyte populations described by Ziegelhoeffer et al.1 Because most dendritic cells are thought to originate from hematopoietic stem cells and to share initial differentiation steps with monocytes,4 we also would like to suggest that further studies on vascular repair should evaluate the presence of specific subpopulations of the monocyte/macrophage cell lineage as well as the role of dendritic cells.1 Ziegelhoeffer T, Fernandez B, Kostin S, Heil M, Voswinckel R, Helisch A, Schaper W. Bone marrow–derived cells do not incorporate into the adult growing vasculature. Circ Res. 2004; 94: 230–238.LinkGoogle Scholar2 Bauriedel G, Jabs A, Skowasch D, Hutter R, Badimon JJ, Fuster A, Welsch U, Lüderitz B. Dendritic cells in neointima formation after rat carotid balloon injury. J Am Coll Cardiol. 2003; 42: 930–938.CrossrefMedlineGoogle Scholar3 Skowasch D, Jabs A, Andrié R, Dinkelbach S, Lüderitz B, Bauriedel G. Presence of bone-marrow- and neural-crest-derived cells in intimal hyperplasia at the time of clinical in-stent restenosis. Cardiovasc Res. 2003; 60: 684–691.CrossrefMedlineGoogle Scholar4 Takahashi T, Lee RT. Dendritic cells in neointima formation: from where did you come, and what are you doing here? J Am Coll Cardiol. 2003; 42: 939–941.CrossrefMedlineGoogle Scholar eLetters(0)eLetters should relate to an article recently published in the journal and are not a forum for providing unpublished data. Comments are reviewed for appropriate use of tone and language. Comments are not peer-reviewed. Acceptable comments are posted to the journal website only. Comments are not published in an issue and are not indexed in PubMed. Comments should be no longer than 500 words and will only be posted online. References are limited to 10. Authors of the article cited in the comment will be invited to reply, as appropriate.Comments and feedback on AHA/ASA Scientific Statements and Guidelines should be directed to the AHA/ASA Manuscript Oversight Committee via its Correspondence page.Sign In to Submit a Response to This Article Previous Back to top Next FiguresReferencesRelatedDetailsCited By Kim J, Jung S, Kwon Y, Lee J, Lee Y, Lee B and Kwon S (2014) Ginsenoside Rg3 attenuates tumor angiogenesis via inhibiting bioactivities of endothelial progenitor cells, Cancer Biology & Therapy, 10.4161/cbt.19599, 13:7, (504-515), Online publication date: 1-May-2012. April 30, 2004Vol 94, Issue 8 Advertisement Article InformationMetrics https://doi.org/10.1161/01.RES.0000126418.31311.F8PMID: 15117831 Originally publishedApril 30, 2004 PDF download Advertisement" @default.
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• W1965816191 title "Bone Marrow–Derived Cells and Vascular Growth" @default.
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