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- W1965980401 abstract "In budding yeast, DNA damage can activate a checkpoint surveillance system controlled by the RAD9, RAD53, and MEC1 genes, resulting in a delay in cell cycle progression. Here, I report that DNA damage induces rapid and extensive phosphorylation of Rad9p in a manner that correlates directly with checkpoint activation. This response is dependent on MEC1, which encodes a member of the evolutionarily conserved ATM family of protein kinases, and on gene products of the RAD24 epistasis group, which have been implicated in the recognition and processing of DNA lesions. Since the phosphorylated form of Rad9p appears capable of interacting stably with Rad53p in vivo, this phosphorylation response likely controls checkpoint signaling by Rad9p." @default.
- W1965980401 created "2016-06-24" @default.
- W1965980401 creator A5076978787 @default.
- W1965980401 date "1998-08-01" @default.
- W1965980401 modified "2023-09-30" @default.
- W1965980401 title "MEC1-Dependent Phosphorylation of Rad9p in Response to DNA Damage" @default.
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- W1965980401 doi "https://doi.org/10.1016/s1097-2765(00)80128-8" @default.
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