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• W1966069708 abstract "The loss of the catabolic products of adenosine triphosphate in the form of purine nucleosides and oxypurines during ischemia and subsequent reperfusion may limit adenine nucleotide regeneration. This study compared the effects of infusion of inhibitors of the major reactions involved in the degradation of adenosine triphosphate to inosine on the postischemic recovery of high energy phosphate and myocardial function. Isolated rat hearts were made totally ischemic after a 5-min infusion of p1, p5-diadenosine pentaphosphate, α,β-methylene adenosine diphosphate, nitrobenzyl-6-thioinosine, or erythro-9-(2-hydroxy-3-nonyl) adenine, which are inhibitors of adenylate kinase, 5′-nucleotidase, adenosine translocase, and adenosine deaminase, respectively. Following 30 min of ischemia, only hearts infused with α,β-methylene adenosine diphosphate recovered significantly better ventricular function than did the control (P < 0.05), but all hearts had increased adenosine triphosphate and creatine phosphate regeneration (P < 0.05). The formation and washout of greater than 30% of the total adenine pool metabolites were not prevented by any drug. Nevertheless all manipulations of adenine metabolism resulted in recruitment of high energy phosphate during preischemic infusion which may have potential benefits in elective ischemic arrest." @default.
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• W1966069708 date "1987-08-01" @default.
• W1966069708 modified "2023-10-14" @default.
• W1966069708 title "Influence of inhibitors of ATP catabolism on myocardial recovery after ischemia" @default.
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• W1966069708 doi "https://doi.org/10.1016/0022-4804(87)90163-6" @default.
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