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- W1966175020 endingPage "1035" @default.
- W1966175020 startingPage "1007" @default.
- W1966175020 abstract "Historically, most drugs developed for treatment of leukemias, lymphomas, and myeloma had already been studied in the solid tumor setting. Nearly 10 years ago, chronic myelogenous leukemia (CML) forever changed this paradigm. Imatinib showed that it was possible to nullify the pathognomic genetic lesion in a hematologic malignancy. Since the approval of imatinib for CML, a host of new drugs active in blood cancers have emerged. This article highlights some areas of innovative drug development in lymphoma where possible; it emphasizes the biologic basis for the approach, linking this essential biology to the biochemical pharmacology. The article focuses on the many new targets including Syk, Bcl-2, CD-40, and the phosphoinositide-3 kinase/AKT/mammalian target of rapamycin pathway." @default.
- W1966175020 created "2016-06-24" @default.
- W1966175020 creator A5002126535 @default.
- W1966175020 creator A5013307217 @default.
- W1966175020 creator A5053153501 @default.
- W1966175020 creator A5062618199 @default.
- W1966175020 creator A5064212964 @default.
- W1966175020 date "2008-10-01" @default.
- W1966175020 modified "2023-09-27" @default.
- W1966175020 title "New Drugs for the Treatment of Lymphoma" @default.
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