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• W1966320394 abstract "The present experiments were undertaken to determine whether the response to nicotine in the isolated canine cerebral artery is endothelium-dependent. Changes in the tension of arterial strips were recorded isometrically. Removal of the endothelium was carried out by gentle rubbing, and confirmed by scanning electron microscopy. Rubbing procedure did not affect the contractile response of the strips to serotonin. Treatment of unrubbed strips with nicotine (10−4M) caused a transient contraction. this response was abolished by removal of endothelium and attenuated by hexamethonium (5 × 10−6M) and atropine (10−6M). The nicotine-induced contraction was attenuated also by aspirin (5 × 10−5M), a cyclo-oxygenase inhibitor, OKY-046 (5 × 10−5M), a thromboxane A2 (TXA2) synthetase inhibitor and ONO-3708 (5 × 10−9M), a TXA 2 antagonist. These results indicate that the nicotine-induced contraction in canine cerebral artery is endothelium-dependent, and suggest that the endothelium-derived contracting factor (EDCF) in the nicotine-induced response is a TXA2-like substance." @default.
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• W1966320394 date "1988-01-01" @default.
• W1966320394 modified "2023-10-15" @default.
• W1966320394 title "Endothelium-dependent contraction induced by nicotine in isolated canine basilar artery -possible involvement of a thromboxane A2 (TXA2) like substance" @default.
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• W1966320394 doi "https://doi.org/10.1016/0024-3205(88)90082-3" @default.
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