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- W1966559621 abstract "The N-terminal region of BRCA2 has the capacity to activate transcription when fused to a heterologous DNA binding domain and includes a segment with amino acid similarity to the JNK-docking site in the cellular JUN protein. However, unlike JUN, we have determined that this region of BRCA2 neither interacts with nor serves as a substrate for JNK, or any other kinase that can be detected in extracts from either fibroblasts or epithelial cells. While this clearly does not rule out a transcriptional role for BRCA2, our findings indicate that BRCA2 is not regulated by the JNK pathway in a manner analogous to JUN. Genes Chromosomes Cancer 25:407–409, 1999. © 1999 Wiley-Liss, Inc." @default.
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- W1966559621 date "1999-08-01" @default.
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- W1966559621 title "The BRCA2 transactivation domain does not interact with JNK" @default.
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- W1966559621 doi "https://doi.org/10.1002/(sici)1098-2264(199908)25:4<407::aid-gcc16>3.0.co;2-i" @default.
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