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• W1967013632 endingPage "630" @default.
• W1967013632 startingPage "625" @default.
• W1967013632 abstract "Oxidative stress has been associated with the onset and progression of mild cognitive impairment (MCI) and Alzheimer disease (AD). AD and MCI brain and plasma display extensive oxidative stress as indexed by protein oxidation, lipid peroxidation, free radical formation, DNA oxidation, and decreased antioxidants. The most abundant endogenous antioxidant, glutathione, plays a significant role in combating oxidative stress. The ratio of oxidized to reduced glutathione is utilized as a measure of intensity of oxidative stress. Antioxidants have long been considered as an approach to slow down AD progression. In this review, we focus on the elevation on glutathione through N-acetyl-cysteine (NAC) and γ-glutamylcysteine ethyl ester (GCEE) as a potential therapeutic approach for Alzheimer disease. This article is part of a Special Issue entitled: Antioxidants and Antioxidant Treatment in Disease." @default.
• W1967013632 created "2016-06-24" @default.
• W1967013632 creator A5004024175 @default.
• W1967013632 creator A5052368753 @default.
• W1967013632 date "2012-05-01" @default.
• W1967013632 modified "2023-10-07" @default.
• W1967013632 title "Elevation of glutathione as a therapeutic strategy in Alzheimer disease" @default.
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• W1967013632 doi "https://doi.org/10.1016/j.bbadis.2011.10.003" @default.
• W1967013632 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/3277671" @default.
• W1967013632 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/22015471" @default.
• W1967013632 hasPublicationYear "2012" @default.
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