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- W1967193457 abstract "To the Editors:A variety of isoforms of nitric oxide (NO) synthases are constitutively expressed in human airway and vascular endothelial cells continuously generating NO. NO plays an important role in regulating lung function in health and disease including modulation of pulmonary vascular resistance, airway calibre and host defence. Production of NO and its consumption by fluid-phase reactions can be detected and monitored in the exhaled air, providing an important window to assess the dynamics of NO metabolism in health and inflammatory lung conditions, asthma in particular 1.A series of milestone studies uncovered a relative deficiency of pulmonary NO availability in cystic fibrosis (CF), a severe chronic inflammatory lung disease with studies generally showing reduced exhaled NO 2, 3. However, there remains controversy regarding the anatomical localisation and mechanisms of NO deficiency, and its relationship with CF disease progression. By using extended exhaled NO analysis at different expiratory flow rates, indirect studies have attempted to distinguish NO production into bronchial and alveolar compartments based on basic models 4–7. However, the fate of NO delivered directly to the alveoli has not been demonstrated in these patients. We aimed to investigate these issues by evaluating endogenous exhaled NO in end-stage CF in intubated patients undergoing lung transplantation and the dynamism of exhaled NO production from pulmonary …" @default.
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- W1967193457 date "2010-08-31" @default.
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- W1967193457 title "Endogenous and nitrovasodilator-induced release of NO in the airways of end-stage cystic fibrosis patients" @default.
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- W1967193457 doi "https://doi.org/10.1183/09031936.00025610" @default.
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