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• W1968650981 abstract "The rise times and half-decay times of extracellularly recorded MEPPs and EPPs decreased almost an order of magnitude during the normal development of the Xenopus myotomal neuromuscular junction. Most of the decrease occurred within the first 18 hr of the life of the synapse. At the earliest stages histochemical staining revealed only weak cholinesterase activity and treatment with the anticholinesterase neostigmine failed to prolong the synaptic potentials. Prolongation of synaptic potentials by neostigmine was observed at later stages and increased with maturation. It is concluded that the development of cholinesterase activity is partially responsible for the developmental shortening of synaptic potentials at this neuromuscular junction. High concentrations of anticholinesterase failed to produce synaptic potentials which were as long as those recorded at the newly formed synapse suggesting that other factors in addition to cholinesterase development, such as changes in receptor channel kinetics or saturation of receptors by transmitter, might also contribute to the developmental change in synaptic potential duration." @default.
• W1968650981 created "2016-06-24" @default.
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• W1968650981 date "1980-03-01" @default.
• W1968650981 modified "2023-09-29" @default.
• W1968650981 title "Contribution of cholinesterase to developmental decreases in the time course of synaptic potentials at an amphibian neuromuscular junction" @default.
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• W1968650981 doi "https://doi.org/10.1016/0012-1606(80)90161-x" @default.
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