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- W1968659112 endingPage "413" @default.
- W1968659112 startingPage "402" @default.
- W1968659112 abstract "Smooth muscle myosin light chain kinase (MLCK) plays a crucial role in artery contraction, which regulates blood pressure and blood flow distribution. In addition to this role, MLCK contributes to Ca(2+) flux regulation in vascular smooth muscle (VSM) and in non-muscle cells, where cytoskeleton has been suggested to help Ca(2+) channels trafficking. This conclusion is based on the use of pharmacological inhibitors of MLCK and molecular and cellular techniques developed to down-regulate the enzyme. Dissimilarities have been observed between cells and whole tissues, as well as between large conductance and small resistance arteries. A differential expression in MLCK and ion channels (either voltage-dependent Ca(2+) channels or non-selective cationic channels) could account for these observations, and is in line with the functional properties of the arteries. A potential involvement of MLCK in the pathways modulating Ca(2+) entry in VSM is described in the present review." @default.
- W1968659112 created "2016-06-24" @default.
- W1968659112 creator A5027983987 @default.
- W1968659112 creator A5043479621 @default.
- W1968659112 creator A5051191638 @default.
- W1968659112 date "2014-09-03" @default.
- W1968659112 modified "2023-10-12" @default.
- W1968659112 title "Regulation of calcium channels in smooth muscle: New insights into the role of myosin light chain kinase" @default.
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