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- W1968881309 abstract "Protective immunity against a Plasmodium falciparum blood infection can be passively transferred by antibodies in humans and in the primate experimental malaria model Saimiri sciureus. We report here the emergence of a novel virulent parasite population after such passive transfer of hyperimmune serum in splenectomized monkeys. These FUP-2 parasites have been partially genotyped and phenotyped. Although no genotypic variation was detected for four polymorphic loci compared to the original FUP-1 parasite population, FUP-2-infected erythrocytes exhibit little or no detectable surface determinants, including those reacting with antibodies raised against FUP-1 surface antigens. In addition, FUP-2-infected erythrocytes exhibit no rosetting or autoagglutination. Interestingly, although Saimiri monkeys control efficiently FUP-2 parasites after repetitive infections, this protection cannot be passively transferred to naive recipients. Our results suggest that antibody-mediated and antibody-independent T-cell-mediated protective responses may cooperate in controlling P. falciparum infection in splenectomized Saimiri monkeys." @default.
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- W1968881309 date "1998-09-01" @default.
- W1968881309 modified "2023-09-27" @default.
- W1968881309 title "Plasmodium falciparum: Immune Pressure inSaimiri sciureusMonkeys Can Select for a Parasite Population Inducing a Protective Immunity That Is Not Controlled by Antibody" @default.
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- W1968881309 doi "https://doi.org/10.1006/expr.1998.4318" @default.
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