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- W1970803670 abstract "Thymosin β4 is a major actin-sequestering molecule. Here, we report a prominent upregulation of thymosin β4 in the hippocampus following entorhinal deafferentation. Northern blotting displayed a transient increase of thymosin β4 mRNA in the deafferented hippocampus by 1.8, 2.3, 1.3 and 1.1-fold of controls, respectively, at 1, 3, 7 and 15 days post-lesion. In-situ hybridization confirmed that the induction of thymosin β4 mRNA specifically occurred in the entorhinally denervated zones of the hippocampus. The double labeling of in-situ hybridization for thymosin β4 mRNA with isolectin B4 cytochemistry showed that isolectin B4-positive microglial cells are responsible for deafferentation-induced thymosin β4 mRNA expression. The results suggest that thymosin β4 may participate in the process of microglial activation, which is the earliest event in lesion-induced plasticity." @default.
- W1970803670 created "2016-06-24" @default.
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- W1970803670 date "2005-10-17" @default.
- W1970803670 modified "2023-09-27" @default.
- W1970803670 title "Expression of thymosin beta4 mRNA by activated microglia in the denervated hippocampus" @default.
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- W1970803670 doi "https://doi.org/10.1097/01.wnr.0000183326.21241.48" @default.
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