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- W1970996854 endingPage "10" @default.
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- W1970996854 abstract "Wound healing involves platelets, inflammatory cells, fibroblasts, and epithelial cells. All of these cell types are capable of producing nitric oxide (NO), either constitutively or in response to inflammatory cytokines, through the activity of nitric oxide synthases (NOSs): eNOS (NOS3; endothelial NOS) and iNOS (NOS2; inducible NOS), respectively. Indeed, pharmacological inhibition or gene deletion of these enzymes impairs wound healing. The wound healing mechanisms that are triggered by NO appear to be diverse, involving inflammation, angiogenesis, and cell proliferation. All of these processes are controlled by defined cytokine cascades; in many cases, NO appears to modulate these cytokines. In this review, we summarize the history and present state of research on the role of NO in wound healing within the framework of modulation of cytokines." @default.
- W1970996854 created "2016-06-24" @default.
- W1970996854 creator A5032978359 @default.
- W1970996854 creator A5062076820 @default.
- W1970996854 creator A5073022985 @default.
- W1970996854 creator A5089042176 @default.
- W1970996854 date "2002-08-01" @default.
- W1970996854 modified "2023-10-17" @default.
- W1970996854 title "Nitric oxide and wound repair: role of cytokines?" @default.
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