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• W1971202273 startingPage "1231" @default.
• W1971202273 abstract "An hypothesis has been presented suggesting that the interaction between cAMP and a steroid hormone may be involved in cellular regulatory mechanisms. The evidence in support of this hypothesis shows that: 1) the interaction between cAMP and estrogen in the growth control of hormone-dependent tumors is antagonistic and requires the respective binding proteins to be present in the cytoplasm; 2) the nuclear translocation of cAMP-binding protein, the regulatory subunit of protein kinase, type II, is inversely related to the nuclear translocation of estrogen receptors during growth and regression of hormone-dependent mammary tumors; 3) the stimulation and prevention of endogenous phosphorylation of nuclear proteins, RAP (regression-associated protein), by cAMP and estrogen in vivo and in vitro, suggest that the counteraction between cAMP and estrogen in growth control may be exerted by a specific action on nuclear protein phosphorylation. Continued investigation on transcriptional control mediated through cAMP and estrogen interaction should provide further insight into the biological regulatory mechanism of hormonal action." @default.
• W1971202273 created "2016-06-24" @default.
• W1971202273 creator A5016789863 @default.
• W1971202273 date "1979-04-01" @default.
• W1971202273 modified "2023-09-26" @default.
• W1971202273 title "On the interaction of cyclic amp-binding protein and estrogen receptor in growth control" @default.
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• W1971202273 doi "https://doi.org/10.1016/0024-3205(79)90142-5" @default.
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• W1971202273 hasPublicationYear "1979" @default.
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