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• W1971377587 abstract "The genesis of pituitary tumours is still under debate. Although these neoplasias are monoclonal in origin, mutations of <i>GNAS1</i>, the gene encoding the α subunit of the stimulatory G-protein, Gs, are the only mutational changes unequivocally associated with growth hormone (GH)-secreting adenomas. However, despite the growth advantage that this oncogene has been demonstrated to confer in vitro, patients carrying this mutation have a similar clinical and biochemical phenotype to those who do not carry it. This discrepancy is due to the occurrence of events able to counteract the biological effect of the mutation. Consistent with a potential role of the cyclic adenosine monophosphate pathway in the proliferation of somatotrophs, germline mutations of the gene encoding the type 1α regulatory subunit of protein kinase A <i>(PRKAR1A)</i> have been found in patients with the Carney complex, a syndrome including GH-secreting adenomas, whereas alterations in the expression levels of this subunit are frequently observed in sporadic adenomas." @default.
• W1971377587 created "2016-06-24" @default.
• W1971377587 creator A5011245991 @default.
• W1971377587 creator A5023054623 @default.
• W1971377587 date "2009-01-01" @default.
• W1971377587 modified "2023-10-17" @default.
• W1971377587 title "G-Protein and Signalling in Pituitary Tumours" @default.
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• W1971377587 doi "https://doi.org/10.1159/000192446" @default.
• W1971377587 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/19407505" @default.
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