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- W1971767825 abstract "Significance Activation of the complement system, a network of circulating and surface-bound molecules, is known to enhance humoral immunity. However, paradoxically, the lack of some complement components (mainly C1q and C4), but not C3, is associated with the development of autoimmunity. Prior studies have suggested that this association could be explained by the role of complement in the disposal of dying cells that are a potential source of autoantigens. This study demonstrates that C3 bound to dying cells can direct the intracellular route of the cargo and modulate the subsequent T-cell response to antigens displayed on dying cells. These results uncover a new role of C3 and have important implications for our understanding of the role of complement in health and disease." @default.
- W1971767825 created "2016-06-24" @default.
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- W1971767825 date "2014-01-13" @default.
- W1971767825 modified "2023-10-17" @default.
- W1971767825 title "C3 opsonization regulates endocytic handling of apoptotic cells resulting in enhanced T-cell responses to cargo-derived antigens" @default.
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- W1971767825 doi "https://doi.org/10.1073/pnas.1316877111" @default.
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