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- W1972065135 abstract "The ability to sense infections is primordial to preserve organisms. Immune cells express pathogen sensors that induct innate and adaptive immune responses. Understanding how HIV-1 infection defeats these responses in most individuals remains an outstanding challenge. Since HIV-1 targets immune cells, innate immune sensors are remarkably positioned at the nexus of viral replication and immunity. Here, we discuss recent studies that have revealed innate sensing mechanisms of HIV-1 infection in plasmacytoid dendritic cells, monocyte-derived dendritic cells, monocyte-derived macrophages, and CD4+ T cells. These studies help understand how HIV-1 avoids antiviral innate immune sensors and how it induces pathogenic processes. Ultimately, this may contribute to therapy and vaccines." @default.
- W1972065135 created "2016-06-24" @default.
- W1972065135 creator A5077609252 @default.
- W1972065135 creator A5087562469 @default.
- W1972065135 date "2015-02-01" @default.
- W1972065135 modified "2023-09-23" @default.
- W1972065135 title "Innate immune sensing of HIV infection" @default.
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- W1972065135 doi "https://doi.org/10.1016/j.coi.2014.12.003" @default.
- W1972065135 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/25617674" @default.
- W1972065135 hasPublicationYear "2015" @default.
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