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- W1972093664 abstract "Several factors, including widespread clinical renal transplantation, histopathologic recognition of glomerular disease within grafts [1–4] and elucidation of the principal immunopathogenetic mechanisms causative of glomerulonephritis in experimental animals and human renal disease [5], have combined to cause keen awareness of the potential risks of recurrent glomerulonephritis in renal grafts. This awareness has been strengthened by reported experience with renal isografts in which most patients whose underlying disease was glomerulonephritis developed glomerulonephritis in their grafts, ultimately causing death in six of the 12 patients affected [6]. Moreover, reports based on immunologic data indicated that pathogenetic autoimmune mechanisms persisting in the host could have important clinical expression in patients with renal allografts, despite immunosuppressive therapy [7]." @default.
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- W1972093664 date "1976-08-01" @default.
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- W1972093664 title "Nephritogenic immunopathologic mechanisms and human renal transplants: The problem of recurrent glomerulonephritis" @default.
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- W1972093664 doi "https://doi.org/10.1038/ki.1976.86" @default.
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