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• W1972364771 abstract "Address for correspondence: Christos Vaklavas, MD, Division of Hematology and Oncology, Department of Medicine, University of Alabama at Birmingham, North Pavillion 2540, 1530 3rd Ave S, Birmingham AL 35294-3300 Clinical Pra ● The prototypic cytogenetic abnormality in acute promyelocytic leukemia (APL) is the reciprocal translocation t(15;17)(q22;q21). ● This leads to the generation of a PML/RAR chimeric protein with dimerization and expanded transcriptional repression abilities. ● Clinical hallmarks of APL constitute an associated hyperfibrinolytic disseminated intravascular coagulation and responsiveness to all-trans retinoic acid (ATRA). ● Cases histologically indistinct from APL without evidence of RAR rearrangement on sensitive molecular analyses are exceedingly rare. ● These cases may harbor alternative cytogenetic abnormalities of possible pathogenetic importance but, rarely, sensitive molecular analyses may be normal. ● We report a case of APL with typical histopathologic features, apparently normal cytogenetic studies, and no evidence of RAR rearrangement on sensitive molecular analyses." @default.
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• W1972364771 date "2011-12-01" @default.
• W1972364771 modified "2023-10-17" @default.
• W1972364771 title "A Case of Acute Promyelocytic Leukemia Without RARα Rearrangement and Apparently Normal Cytogenetics" @default.
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• W1972364771 doi "https://doi.org/10.1016/j.clml.2011.03.036" @default.
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