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- W1972603676 endingPage "944" @default.
- W1972603676 startingPage "936" @default.
- W1972603676 abstract "Genomic DNA is particularly vulnerable to mutation during S-phase when the two strands of parental duplex DNA are separated during the process of semi-conservative DNA replication. Lesions that are normally repaired efficiently in the context of double stranded DNA can cause replication forks to stall or, more dangerously, collapse. Cells from Bloom's syndrome patients, that lack the RecQ helicase BLM, show defects in the response to replicative stress and contain a multitude of chromosomal aberrations, which primarily arise through excessive levels of homologous recombination. Here, recent findings are reviewed that further our understanding of the role that BLM plays in the management of damaged replication forks." @default.
- W1972603676 created "2016-06-24" @default.
- W1972603676 creator A5049974556 @default.
- W1972603676 date "2007-07-01" @default.
- W1972603676 modified "2023-09-23" @default.
- W1972603676 title "Role of the BLM helicase in replication fork management" @default.
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