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• W1972741570 abstract "1 Continuous recording of cardiac contractions and coronary flow from isolated perfused hearts of rats permitted the study of coronary reactions to: (a) cardiostimulation induced by single doses or slow infusions of noradrenaline, CaCl2, glucagon or electrically induced tachycardia; (b) short interruptions of coronary inflow (hypoxia). 2 Except during tachycardia the heart rate was kept constant at 210 beats/min by electrical pacing. 3 Metabolic coronary vasodilatation (MCD) resulting from cardiac hyperactivity induced by noradrenaline, Ca2+, tachycardia or glucagon was inhibited by administration of prostaglandin E2. Reactive hyperaemia response to hypoxia was unaffected by prostaglandin administration. 4 Inhibition of MCD could also be obtained by prolonged infusion with arachidonic acid (1.6 X 10(-7) M), presumably by its conversion into prostaglandin-like substance since arachidonic acid failed to block MCD in hearts from rats pretreated with non-steroidal anti-inflammatory drugs (indomethacin, naproxen, phenylbutazone). 5 Reactive hyperaemia was unaffected either by arachidonic acid or by blockade of the synthesis of prostaglandin-like substances by anti-inflammatory drugs. 6 Since prostaglandin synthetase inhibition does not prevent but may enhance MCD, we do not advocate prostaglandin-like substances as agents directly responsible for the coronary vasodilatation that follows cardiac hyperactivity. 7 We postulate that cardiac overproduction of prostaglandins may lead to a failure in the adaptive coronary flow response to cardiac hyperactivity (coronary insufficiency?)." @default.
• W1972741570 created "2016-06-24" @default.
• W1972741570 creator A5019034589 @default.
• W1972741570 creator A5087743954 @default.
• W1972741570 date "1979-01-01" @default.
• W1972741570 modified "2023-10-18" @default.
• W1972741570 title "MYOCARDIAL SYNTHESIS OF PROSTAGLANDIN-LIKE SUBSTANCES AND CORONARY REACTIONS TO CARDIOSTIMULATION AND TO HYPOXIA" @default.
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• W1972741570 doi "https://doi.org/10.1111/j.1476-5381.1979.tb17335.x" @default.
• W1972741570 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/1668475" @default.
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