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• W197311864 abstract "This chapter describes the changes of neuronal function and other physiological parameters. A common carotid artery (CCA) occlusion in Mongolian gerbils, as well as regional differences of these parameters is also described. In a method described in the chapter, bilateral CCA occlusion produces severe ischemia in both cortex and hippocampus. During CCA occlusion, the forebrain reveals severe ischemia followed by heterogeneous hyperemia after the occlusion is released. The chapter presents the morphological observations that reveal that five minutes of forebrain ischemia in gerbils lead to highly reproducible delayed destruction of neurons in the hippocampal CA1 subfield. With respect to CAlN, notable changes are observed first at two days following ischemia, at the time neurons show some loss of Nissl substance, a more accentuated staining of dendrites, and appearance of clear clefts in the cytoplasm. After three days, most of the CAlN shows severe ischemic injury, followed by neuronal disintegration at four days. The cortex showed no pathological changes during the whole observation period. The described changes of various parameters resulting from five minutes ischemia in gerbils demonstrate that ischemic death of CAlN takes place during the recirculation period." @default.
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• W197311864 date "1985-01-01" @default.
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• W197311864 title "Microphysiology of Selectively Vulnerable Neurons" @default.
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• W197311864 doi "https://doi.org/10.1016/s0079-6123(08)61975-5" @default.
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