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- W1973996890 abstract "The alternative pathway of complement is implicated in the pathogenesis of several renal diseases, such as atypical hemolytic uremic syndrome, dense deposit disease and other forms of C3 glomerulopathy. The underlying complement defects include genetic and/or acquired factors, the latter in the form of autoantibodies. Because the autoimmune forms require a specific treatment, in part different from that of the genetic forms, it is important to detect the autoantibodies as soon as possible and understand their characteristics. In this overview, we summarize the types of anti-complement autoantibodies detected in such diseases, i.e. autoantibodies to factor H, factor I, C3b, factor B and those against the C3 convertases (C3 nephritic factor and C4 nephritic factor). We draw attention to newly described autoantibodies and their characteristics, and highlight similarities and differences in the autoimmune forms of these diseases." @default.
- W1973996890 created "2016-06-24" @default.
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- W1973996890 creator A5031942816 @default.
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- W1973996890 date "2014-08-01" @default.
- W1973996890 modified "2023-10-06" @default.
- W1973996890 title "Autoantibodies to complement components in C3 glomerulopathy and atypical hemolytic uremic syndrome" @default.
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- W1973996890 doi "https://doi.org/10.1016/j.imlet.2014.01.014" @default.
- W1973996890 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/24491679" @default.
- W1973996890 hasPublicationYear "2014" @default.
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