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• W1974041167 abstract "Bilateral destruction of the hypothalamic paraventricular nuclei (PVN) produced a profound depression of plasma TSH and the median eminence TRH concentration in hypothyroid rats. Anterior pituitary type II iodothyronine 5′- deiodinase (5′-D) activity was consistently lower but not significantly different in sham- and PVN-lesioned rats. Treatment with suboptimal replacement doses of 0.15 and 0.75 ng T4/100 g BW-day produced a graded depression of plasma TSH in the PVN (P < 0.02), but not in the sham (P > 0.8) groups. Adenohypophyseal 5′-D was depressed in both sham and PVN groups by the highest T4 dose. Plasma T4 was much lower in PVN than in sham rats given comparable doses of T4 (P < 0.001), but plasma T3 was not significantly different. This suggests that an increase in peripheral T4 metabolism was produced by PVN lesions. Our data indicate that changes in adenohypophyseal 5′- D activity are not responsible for the decrease in plasma TSH in PVN-lesioned rats and that neither the PVN nor endogenous TRH plays a significant role in the regulation of anterior pituitary 5′-D activity. (Endocrinology123: 1676–1681, 1988)" @default.
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• W1974041167 date "1988-09-01" @default.
• W1974041167 modified "2023-09-26" @default.
• W1974041167 title "Anterior Pituitary Type II Thyroxine 5′-Deiodinase Activity is not Affected by Lesions of the Hypothalamic Paraventricular Nucleus which Profoundly Depress Pituitary Thyrotropin Secretion*" @default.
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• W1974041167 doi "https://doi.org/10.1210/endo-123-3-1676" @default.
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