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• W1974804971 abstract "When [14C]histamine was administered orally to rats, an average of 80% of the administered radioactivity was recovered in the urine at the end of 24 hr. About 10% of the total dose was excreted via the feces. Analysis of 4-hr urine samples found imidazoleacetic acid to be the predominant metabolite (60.6%), with Nτ-methylimidazoleacetic acid (8.6%), Nτ-methylhistamine (7.3%), and N-acetylhistamine (4.5%) to be the minor metabolites. Histamine metabolism was inhibited by simultaneous oral administration of aminoguanidine, isoniazid, quinacrine, cadaverine, putrescine, tyramine, and β-phenylethylamine. The administration of inhibitors resulted in an increased amount of unmetabolized histamine and a decreased amount of metabolites reaching the urine. Pharmacologic inhibitors were found to be more potent and have a longer duration of action than foodborne ones. The inhibitors could potentiate food poisoning caused by histamine by inhibiting its metabolism." @default.
• W1974804971 created "2016-06-24" @default.
• W1974804971 creator A5017425415 @default.
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• W1974804971 date "1985-11-01" @default.
• W1974804971 modified "2023-10-14" @default.
• W1974804971 title "Inhibition of in vivo histamine metabolism in rats by foodborne and pharmacologic inhibitors of diamine oxidase, histamine N-methyltransferase, and monoamine oxidase" @default.
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• W1974804971 doi "https://doi.org/10.1016/0041-008x(85)90160-7" @default.
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