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- W1976670388 endingPage "442" @default.
- W1976670388 startingPage "427" @default.
- W1976670388 abstract "Background: There is increasing evidence that Toll-like receptors (TLRs) sense host tissue damage by engaging with endogenous ligands. TLRs are considered to be involved in many primarily non-immune-related diseases. Hepatic ischemia reperfusion injury (IRI) represents one of these disorders. Objective: To present the latest findings supporting the involvement of TLRs in liver IRI and to explore their role as potential targets for therapeutic intervention. Methods: A review of the literature summarizing the latest advances in TLR signaling, the role of TLRs in each hepatic cell population and the involvement of TLRs in the pathophysiology of hepatic IRI. The potential role of TLR-targeting treatment strategies in liver IRI is discussed. Conclusions: Recent experimental evidence suggests that TLR activation on Kupffer cells provides the triggering signal for pro-inflammatory responses that lead to liver IRI. Modulating TLR signaling could have a beneficial effect in patients with liver IRI." @default.
- W1976670388 created "2016-06-24" @default.
- W1976670388 creator A5006412575 @default.
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- W1976670388 creator A5072192151 @default.
- W1976670388 creator A5076360091 @default.
- W1976670388 date "2009-03-31" @default.
- W1976670388 modified "2023-10-18" @default.
- W1976670388 title "Toll like receptors in liver ischemia reperfusion injury: A novel target for therapeutic modulation?" @default.
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