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- W1976804963 endingPage "743" @default.
- W1976804963 startingPage "734" @default.
- W1976804963 abstract "Atrial fibrillation (AF) is the most common cardiac arrhythmia and is associated with substantial morbidity and mortality. It causes profound changes in sarcoplasmic reticulum (SR) Ca2+ homeostasis, including ryanodine receptor channel dysfunction and diastolic SR Ca2+ leak, which might contribute to both decreased contractile function and increased propensity to atrial arrhythmias. In this review, we will focus on the molecular basis of ryanodine receptor channel dysfunction and enhanced diastolic SR Ca2+ leak in AF. The potential relevance of increased incidence of spontaneous SR Ca2+ release for both AF induction and/or maintenance and the development of novel mechanism-based therapeutic approaches will be discussed." @default.
- W1976804963 created "2016-06-24" @default.
- W1976804963 creator A5000609088 @default.
- W1976804963 creator A5027161261 @default.
- W1976804963 creator A5028184651 @default.
- W1976804963 date "2010-10-12" @default.
- W1976804963 modified "2023-10-15" @default.
- W1976804963 title "The ryanodine receptor channel as a molecular motif in atrial fibrillation: pathophysiological and therapeutic implications" @default.
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