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• W1977687670 endingPage "264" @default.
• W1977687670 startingPage "251" @default.
• W1977687670 abstract "We have replaced the Hox-3.1 coding sequence with the E. coli lacZ gene by means of homologous recombination in embryonic stem cells and thus produced null mutant mice. Homozygous mice were born alive, but most of them died within a few days. In the trunk region of homozygotes, several skeletal segments were transformed into the likeness of more anterior ones, as observed in Drosophila with loss-of-function homeotic mutations. The most obvious transformations were the attachment of the 8th pair of ribs to the sternum and the appearance of a 14th pair of ribs on the 1st lumbar vertebra. The pattern of beta-galactosidase activity was identical in heterozygotes and homozygotes and reflected faithfully the Hox-3.1 expression pattern. Thus, the mutation modified the identity, rather than the position, of embryonic cells that would normally express Hox-3.1." @default.
• W1977687670 created "2016-06-24" @default.
• W1977687670 creator A5074658493 @default.
• W1977687670 creator A5075767866 @default.
• W1977687670 creator A5083607278 @default.
• W1977687670 date "1992-04-01" @default.
• W1977687670 modified "2023-09-27" @default.
• W1977687670 title "Homeosis in the mouse induced by a null mutation in the Hox-3.1 gene" @default.
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• W1977687670 doi "https://doi.org/10.1016/0092-8674(92)90406-3" @default.
• W1977687670 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/1348969" @default.
• W1977687670 hasPublicationYear "1992" @default.
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