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- W1978016611 endingPage "186" @default.
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- W1978016611 abstract "Osteoclasts are bone-resorbing cells essential for skeletal remodeling and regeneration. However, excessive osteoclasts often contribute to prevalent bone degenerative diseases such as osteoporosis, arthritis, and cancer bone metastasis. Osteoclast dysregulation is also associated with rare disorders such as osteopetrosis, pycnodysostosis, Paget's disease, and Gorham-Stout syndrome. The nuclear receptor (NR) family of transcription factors functions as metabolic sensors that control a variety of physiological processes including skeletal homeostasis and serves as attractive therapeutic targets for many diseases. In this review, we highlight recent findings on the new players and the new mechanisms for how NRs regulate osteoclast differentiation and bone resorption. An enhanced understanding of NR functions in osteoclastogenesis will facilitate the development of not only novel osteoprotective medicine but also prudent strategies to minimize the adverse skeletal effects of certain NR-targeting drugs for a better treatment of cancer and metabolic diseases." @default.
- W1978016611 created "2016-06-24" @default.
- W1978016611 creator A5074972238 @default.
- W1978016611 creator A5084136148 @default.
- W1978016611 creator A5088533080 @default.
- W1978016611 date "2015-02-01" @default.
- W1978016611 modified "2023-09-28" @default.
- W1978016611 title "Minireview: Nuclear Receptor Regulation of Osteoclast and Bone Remodeling" @default.
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