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• W1978110174 abstract "In addition to the well established cardiovascular risk factors, evidence suggests a possible role of genetic and non-classical risk factors in the development and progression of atherothrombosis. We aimed to determine the relationship of classical and non-classical cardiovascular risk factors with candidate gene polymorphisms potentially involved in cardiovascular risk in the general Mediterranean population. Cross-sectional study. We have determined the prevalence of classical (lipid profile, blood pressure, glycaemia, diabetes, smoking, body mass index, menopause and family history of coronary heart disease) and non-classical cardiovascular risk factors (infectious processes, homocysteinaemia, oxidative status, C-reactive protein, lipoprotein (a) and fibrinogen) in a population-based study. We analysed the relationship of these risk factors with the following five gene polymorphisms potentially involved in cardiovascular risk: ATP-binding cassette transporter A1-R219K, Peroxisome proliferator-activated receptor (PPAR)-α-L162V, Lipoprotein lipase (LPL)–HindIII, Paraoxonase (PON)1-Q192R, and Tumour necrosis factor (TNF)-α- G-308A. We found PPAR-α-V and LPL-H+ alleles to be associated with decreased high-density lipoprotein-cholesterol (HDL-c) concentration and with increased total cholesterol: HDL-c and triglyceride: HDL-c ratios. Regarding the non-classical risk factors, C-reactive protein concentration was higher for the PPAR-α-V allele. A higher oxidative status was shown in homozygotes for LPL-H+ and TNF-α-G alleles, although the latter also had lower homocysteinaemia. Three of the genetic variants analysed, PPAR-α-L162V, LPL-HindIII, and TNF-α-G-308A, were associated with non-classical risk factors, specifically lipid profile, inflammation, and oxidative status." @default.
• W1978110174 created "2016-06-24" @default.
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• W1978110174 date "2006-10-01" @default.
• W1978110174 modified "2023-10-18" @default.
• W1978110174 title "Relationship of classical and non-classical risk factors with genetic variants relevant to coronary heart disease" @default.
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• W1978110174 doi "https://doi.org/10.1097/01.hjr.0000224484.80349.3f" @default.
• W1978110174 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/17001213" @default.
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