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- W1978392572 abstract "The loss of vision in the human eye disease, glaucoma, is due to degeneration of the axons of the retinal ganglion cells. In glaucoma, reactive astrocytes in the optic nerve head contain inducible nitric oxide synthase, which apparently produces excessive nitric oxide that damages the axons. The astrocytes respond to the elevated intraocular pressure that is characteristic of the disease. An important signal transduction pathway for the induction of nitric oxide synthase in response to pressure is the epidermal growth factor receptor tyrosine kinase. Pharmacological inhibition of the activity or the induction of inducible nitric oxide synthase may provide neuroprotection for the treatment of glaucoma." @default.
- W1978392572 created "2016-06-24" @default.
- W1978392572 creator A5048826252 @default.
- W1978392572 creator A5072482442 @default.
- W1978392572 date "2003-12-01" @default.
- W1978392572 modified "2023-10-04" @default.
- W1978392572 title "Glaucomatous Optic Neuropathy: When Glia Misbehave" @default.
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- W1978392572 doi "https://doi.org/10.1177/1073858403253460" @default.
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