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• W1978646572 abstract "Neurotrophins are involved in the modulation of synaptic transmission, including the induction of long-term potentiation (LTP) through the receptor TrkB. Because previous studies have revealed a bidirectional mode of neurotrophin action by virtue of signaling through either the neurotrophin receptor p75NTR or the Trk receptors, we tested the hypothesis that p75NTR is important for longterm depression (LTD) to occur. Although LTP was found to be unaffected in hippocampal slices of two different strains of mice carrying mutations of the p75NTR gene, hippocampal LTD was impaired in both p75NTR-deficient mouse strains. Furthermore, the expression levels of two (RS)-alpha-amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA) receptor subunits, GluR2 and GluR3, but not GluR1 or GluR4, were found to be significantly altered in the hippocampus of p75NTR-deficient mice. These results implicate p75NTR in activity-dependent synaptic plasticity and extend the concept of functional antagonism of the neurotrophin signaling system." @default.
• W1978646572 created "2016-06-24" @default.
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• W1978646572 date "2005-05-09" @default.
• W1978646572 modified "2023-10-09" @default.
• W1978646572 title "The neurotrophin receptor p75 ^NTR modulates long-term depression and regulates the expression of AMPA receptor subunits in the hippocampus" @default.
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• W1978646572 doi "https://doi.org/10.1073/pnas.0502460102" @default.
• W1978646572 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/1129133" @default.
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