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- W1978709703 abstract "Pulmonary artery hypertension (PAH) is a proliferative disorder associated with enhanced pulmonary artery smooth muscle cell proliferation and suppressed apoptosis. The sustainability of this phenotype requires the activation of pro-survival transcription factor like the signal transducers and activators of transcription-3 (STAT3). Using multidisciplinary and translational approaches, we and others have demonstrated that STAT3 activation in both human and experimental models of PAH accounts for the modulation of the expression of several proteins already known as implicated in PAH pathogenesis, as well as for signal transduction to other transcription factors. Furthermore, recent data demonstrated that STAT3 could be therapeutically targeted in different animal models and some molecules are actually in clinical trials for cancer or PAH treatment." @default.
- W1978709703 created "2016-06-24" @default.
- W1978709703 creator A5026341380 @default.
- W1978709703 creator A5045468605 @default.
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- W1978709703 date "2012-10-01" @default.
- W1978709703 modified "2023-10-17" @default.
- W1978709703 title "STAT3 signaling in pulmonary arterial hypertension" @default.
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