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- W1978904983 abstract "Diabetes mellitus is an important risk factor of periodontal diseases. It is one of the main contributing factors for periodontal disease and also limiting factor for periodontal treatments such as implant therapy. Although diabetes itself does not cause periodontitis, periodontal disease progresses more rapidly and leads to more tooth losses in patients with poorly controlled blood glucose. Severe periodontitis has been associated with an increased risk of poor glycemic control and, in turn untreated advanced periodontal disease can deteriorate the metabolic control of diabetes. Various pathogenetic factors have been suggested to explain the increased prevalence and severity of periodontitis in diabetes.1) Reduced polymorphonuclear leukocyte(PMN) function has been found in patients with diabetes. This impairment of function was noted in assays of PMN chemotaxis, adherence and phagocytosis. Studies of PMN defects suggest that this dysfunction could lead to impaired host resistance to infection. Gingival fibroblasts from diabetic patients synthesize less collagen compared to non-diabetic subjects. In addition to decreased collagen production, crevicular fluid collagenolytic activity also was increased in diabetic patients. This increased crevicular fluid collagenase activity appears to be primarily of neutrophil origin. Non-enzymatic glycosylation process results in increased cross-linking between collagen molecules. This cross-linking of collagen significantly contributes to reduced solubility and de-" @default.
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- W1978904983 date "2007-01-01" @default.
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- W1978904983 title "The comparison of IL-6, elastase and α-PI expressions in human chronic periodontitis with type 2 diabetes mellitus" @default.
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- W1978904983 doi "https://doi.org/10.5051/jkape.2007.37.suppl.325" @default.
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