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• W1979047904 abstract "<h2>Abstract</h2> This study examines the role of Th1 (interferon-gamma [IFN-γ]) and Th2 (interleukin-4 [IL-4] and IL-10) cytokines, an intercellular adhesion molecule (ICAM-1), and a chemokine receptor (CCR5) in the pathogenesis of periapical lesions at different stages of development in knockout mice. For lesion induction, the first molar was opened and inoculated with 4 bacterial strains and left open to the oral environment. After 21 and 42 days, the IFN-γ, IL-10, ICAM-1, and CCR5 knockout animals presented periapical lesions larger than those of wild-type animals. There was no statistically significant difference between periapical lesions induced in IL-4 knockout and wild-type animals during the periods evaluated. Our findings suggest an important role for IFN-γ, IL-10, ICAM-1, and CCR5 in the pathogenesis of experimentally induced pulp infection and bone destruction as endogenous suppressor of periapical lesion development, whereas IL-4 appears to present a nonsignificant effect on periapical lesion modulation." @default.
• W1979047904 created "2016-06-24" @default.
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• W1979047904 date "2008-01-01" @default.
• W1979047904 modified "2023-09-26" @default.
• W1979047904 title "Interferon-gamma, Interleukin-10, Intercellular Adhesion Molecule-1, and Chemokine Receptor 5, but not Interleukin-4, Attenuate the Development of Periapical Lesions" @default.
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• W1979047904 doi "https://doi.org/10.1016/j.joen.2007.09.021" @default.
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