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- W1979183749 abstract "Using Rbp4-null mice as models, we have established for the first time the kinetics of the spermatogenetic alterations during vitamin A deficiency (VAD). Our data demonstrate that the VAD-induced testicular degeneration arises through the normal maturation of germ cells in a context of spermatogonia differentiation arrest. They indicate that retinoic acid (RA) appears dispensable for the transition of premeiotic to meiotic spermatocytes, meiosis, and spermiogenesis. They confirm that RA plays critical roles in controlling spermatogonia differentiation, spermatid adhesion to Sertoli cells, and spermiation, and suggest that the VAD-induced arrest of spermatogonia differentiation results from simultaneous blocks in RA-dependent events mediated by RA receptor γ (RARγ) in spermatogonia and by RARα in Sertoli cells. They also provide evidence that expression of major RA-metabolizing enzymes is increased in mouse Sertoli cells upon VAD and that vitamin A-deficient A spermatogonia differ from their RA-sufficient counterparts by the expression of the Stra8 gene. Developmental Dynamics 235:1608–1622, 2006. © 2006 Wiley-Liss, Inc." @default.
- W1979183749 created "2016-06-24" @default.
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- W1979183749 date "2006-06-01" @default.
- W1979183749 modified "2023-10-02" @default.
- W1979183749 title "Retinoids and spermatogenesis: Lessons from mutant mice lacking the plasma retinol binding protein" @default.
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- W1979183749 doi "https://doi.org/10.1002/dvdy.20795" @default.
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