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• W1979184108 abstract "Parathyroid hormone (PTH) promotes IL-6 secretion by osteoblasts, and may also up-regulate IL-6 production in the liver and adipose tissue; this may explain why serum IL-6 is markedly elevated in primary hyperparathyroidism, and low in hypoparathyroidism. IL-6 is the chief stimulus to hepatic production of many acute phase reactants, notably fibrinogen and C-reactive protein (CRP). Mild secondary hyperparathyroidism is common in elderly people, particularly at high latitudes during the winter, owing to poor vitamin D status. This may rationalize evidence that acute phase proteins show seasonal variations and are typically elevated in the elderly, whereas leisure physical activity is associated with a reduction in these proteins. In a recent clinical trial targeting elderly chronically ill patients, administration of vitamin D reduced serum levels of both CRP and IL-6; further such studies should assess the impact of physiologically meaningful doses of vitamin D on acute phase reactants in elderly subjects likely to have poor vitamin D status. Since elevations of CRP and fibrinogen may increase risk for thromboembolic vascular events, these considerations may help to explain the excess of coronary mortality observed during winter months, and suggest a role for supplemental vitamin D in preservation of vascular health. Moderate alcohol intake is associated with reduced serum PTH as well as decreased levels of CRP and fibrinogen; conceivably, modulation of PTH mediates, at least in part, the favorable impact of moderate drinking on the acute phase reactants." @default.
• W1979184108 created "2016-06-24" @default.
• W1979184108 creator A5064469464 @default.
• W1979184108 date "2005-01-01" @default.
• W1979184108 modified "2023-09-25" @default.
• W1979184108 title "Secondary hyperparathyroidism promotes the acute phase response – a rationale for supplemental Vitamin D in prevention of vascular events in the elderly" @default.
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• W1979184108 doi "https://doi.org/10.1016/j.mehy.2004.03.041" @default.
• W1979184108 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/15780504" @default.
• W1979184108 hasPublicationYear "2005" @default.
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