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• W1980866580 abstract "Background We assumed that increased mast cell numbers contribute substantially to the fibrosis often seen in the peritoneum of peritoneal dialysis (PD) patients, particularly those with encapsulating peritoneal fibrosis (EPS). Therefore, we investigated mast cells in different pathological conditions of the peritoneum. Methods One hundred fifteen tissue probes with different peritoneal pathological states were selected (normal, n = 20; chronic appendicitis, n = 25; herniotomy, n = 24; fibrosis, n = 11; PD, n = 26; and EPS, n = 9). For staining of mast cells, we used α-naphtol-AS-d-chloracetate-esterase and mast cell tryptase. Next, we counted numbers of mast cells per square millimeter. Tryptase was measured by using image analysis. Results Measurements by means of both methods correlated well (r = 0.812). Numbers of mast cells per square millimeter were as follows: normal, 26 ± 16; chronic appendicitis, 241 ± 217; herniotomy, 115 ± 88; fibrosis, 99 ± 66; PD, 81 ± 64, and EPS, 24 ± 23 (P = 0.00006). Amounts of tryptase present were 2.900 ± 0.118, 2.871 ± 0.150, 2.733 ± 0.183, 3.041 ± 0.176, 2.780 ± 0.184, and 2.609 ± 0.234, respectively (P = 0.00002). Conclusion We found upregulation of mast cells in specimens of chronic inflammatory diseases of the peritoneum. This also was true for PD patients, with the exclusion of patients with EPS. Therefore, loss-of-control functions of mast cells may contribute to the ill-understood disease entity of EPS. We assumed that increased mast cell numbers contribute substantially to the fibrosis often seen in the peritoneum of peritoneal dialysis (PD) patients, particularly those with encapsulating peritoneal fibrosis (EPS). Therefore, we investigated mast cells in different pathological conditions of the peritoneum. One hundred fifteen tissue probes with different peritoneal pathological states were selected (normal, n = 20; chronic appendicitis, n = 25; herniotomy, n = 24; fibrosis, n = 11; PD, n = 26; and EPS, n = 9). For staining of mast cells, we used α-naphtol-AS-d-chloracetate-esterase and mast cell tryptase. Next, we counted numbers of mast cells per square millimeter. Tryptase was measured by using image analysis. Measurements by means of both methods correlated well (r = 0.812). Numbers of mast cells per square millimeter were as follows: normal, 26 ± 16; chronic appendicitis, 241 ± 217; herniotomy, 115 ± 88; fibrosis, 99 ± 66; PD, 81 ± 64, and EPS, 24 ± 23 (P = 0.00006). Amounts of tryptase present were 2.900 ± 0.118, 2.871 ± 0.150, 2.733 ± 0.183, 3.041 ± 0.176, 2.780 ± 0.184, and 2.609 ± 0.234, respectively (P = 0.00002). We found upregulation of mast cells in specimens of chronic inflammatory diseases of the peritoneum. This also was true for PD patients, with the exclusion of patients with EPS. Therefore, loss-of-control functions of mast cells may contribute to the ill-understood disease entity of EPS." @default.
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• W1980866580 date "2007-03-01" @default.
• W1980866580 modified "2023-10-18" @default.
• W1980866580 title "Peritoneal Mast Cells in Peritoneal Dialysis Patients, Particularly in Encapsulating Peritoneal Sclerosis Patients" @default.
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• W1980866580 doi "https://doi.org/10.1053/j.ajkd.2006.11.040" @default.
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