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• W1981011630 abstract "Ethanol has central serotonergic effects that may be of pathogenetic importance in a subgroup of alcohol-dependent patients with a central serotonergic hypofunction. Recent results indicate that pronounced amplitude increases of auditory evoked responses (tangential dipoles, N1/P2 component) with increasing stimulus intensity (loudness) may be an indicator of such a low serotonergic neurotransmission. Because of its serotonin-agonistic effects, ethanol can be expected to decrease this intensity dependence. Twenty-eight alcoholic patients were studied both in the intoxication phase and after 1 week of withdrawal. A reduced intensity dependence of the tangential dipole activity was observed in the intoxicated state. Correspondingly, a reduction of this parameter was found in 14 healthy subjects after an ethanol load (1 g/kg, p.o.)." @default.
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• W1981011630 date "1996-06-01" @default.
• W1981011630 modified "2023-10-18" @default.
• W1981011630 title "Serotonergic ethanol effects and auditory evoked dipole activity in alcoholic and healthy subjects" @default.
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• W1981011630 doi "https://doi.org/10.1016/0165-1781(96)02796-5" @default.
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