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- W1981054311 abstract "Abstract One of the responses to cerebral ischemia is an increase in the production of nitric oxide, catalyzed by enzymes expressed in both resident and infiltrating cells. The nitric oxide that is generated does contribute to the ensuing pathology, but it can also be beneficial. The effects of nitric oxide depend on the cell site of production, the amount generated, and the chemical nature of the products of further oxidation. Understanding how nitric oxide production from microglia and astrocytes contributes to ischemic pathology is important for the development and application of future therapeutics based on inhibiting or amplifying its production in the injured brain. © 2005 Wiley‐Liss, Inc." @default.
- W1981054311 created "2016-06-24" @default.
- W1981054311 creator A5024187061 @default.
- W1981054311 creator A5052634118 @default.
- W1981054311 creator A5061053285 @default.
- W1981054311 date "2005-04-21" @default.
- W1981054311 modified "2023-10-18" @default.
- W1981054311 title "Glial nitric oxide and ischemia" @default.
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